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SRX23443816: GSM8045617: vancomcycin_30mins_3; Staphylococcus aureus subsp. aureus str. JKD6008; RNA-Seq
1 ILLUMINA (Illumina NovaSeq 6000) run: 12.2M spots, 3.7G bases, 1.1Gb downloads

External Id: GSM8045617_r1
Submitted by: Tree lab, School of Biotechnology and Biomolecular Sciences, University of New South Wales
Study: The functional small RNA interactome reveals targets for the vancomycin-responsive sRNA RsaOI in vancomycin tolerant Staphylococcus aureus [RNA-seq]
show Abstracthide Abstract
Small RNAs have been found to control a broad range of bacterial phenotypes including tolerance to antibiotics. Vancomycin tolerance in multidrug resistance Staphylococcus aureus is correlated with dysregulation of small RNAs although their contribution to antibiotic tolerance in poorly understood. RNA-RNA interactome profiling techniques are expanding our understanding of sRNA-mRNA interactions in bacteria; however, determining the function of these interactions for hundreds of sRNA-mRNA pairs is a major challenge. At steady-state, protein and mRNA abundances are often highly correlated and lower than expected protein abundance may indicate translational repression of an mRNA. To identify sRNA-mRNA interactions that regulate mRNA translation, we examined the correlation between gene transcript abundance, ribosome occupancy, and protein levels. We used the machine learning technique self-organising maps (SOMS) to cluster genes with similar transcription and translation patterns and identified a cluster of mRNAs that appeared to be post-transcriptionally repressed. By integrating our clustering with sRNA-mRNA interactome data generated in vancomycin tolerant S. aureus by RNase III-CLASH, we identified sRNAs that may be mediating translational repression. We have confirmed sRNA-dependant post-transcriptional repression of several mRNAs in this cluster. Two of these interactions are mediated by RsaOI, a sRNA that is highly upregulated by vancomycin. We demonstrate regulation of HPr and the cell-wall autolysin Atl. These findings suggest RsaOI coordinates carbon metabolism and cell wall turnover during vancomycin treatment. Overall design: This study uses a metaomics approach to assess gene transcription and translation, and identify genes that are post-transcriptionally regulated. RNA-seq is used to assess mRNA abundance and is presented in this entry. Total RNA was extracted from S. aureus strain JKD6008 (VISA) in biological triplicate. Cultures were prepared with or without treatment with 8ug/ml vancomycin for 30 minutes.
Sample: vancomcycin_30mins_3
SAMN39667161 • SRS20297173 • All experiments • All runs
Library:
Name: GSM8045617
Instrument: Illumina NovaSeq 6000
Strategy: RNA-Seq
Source: TRANSCRIPTOMIC
Selection: cDNA
Layout: PAIRED
Construction protocol: GTF-phenol (Tollervey & Mattaj EMBO J 1987; PMID: 2953599) Libraries prepared by Novagene using prokaryotic directional mRNA library preparation (with rRNA removal).
Runs: 1 run, 12.2M spots, 3.7G bases, 1.1Gb
Run# of Spots# of BasesSizePublished
SRR2777891212,214,2783.7G1.1Gb2024-03-18

ID:
31674978

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